Monday, August 4, 2014

How Chlorophyll Can Help Eliminate Bad Breath

How Chlorophyll Can Help Eliminate Bad Breath
Chlorophyll is the green pigment in plants that give most their
color and enables them to carry on the process of photosynthesis.
Chlorophyll, found in the chloroplasts of the plant cell, is the only
substance in nature able to trap and store the energy of sunlight.



Chlorophyll helps to remove body odour, eliminate bad breath,
relieve sore throat, improve blood circulation, reduce indigestion and
reduce tiredness. Among its other virtues, Chlorophyll apparently makes
cells more resistant to bacteria, stimulates wound healing, helps in
the treatment of peptic ulcers, and promotes bowel regularity.
Chlorophyll, an alkalinizing food, can help offset the acidifying
effect of the typical high-fat, high-protein Western diet.


Chlorophyll, the green pigments in plants, is remarkably similar in
molecular structure to hemoglobin, a protein in the human red blood
cells that carries oxygen to bodily tissues. One reason for their
beneficial effects on your health is, chlorophyll reduces the acidity
in your body: Stress, and too much protein and fat in your diet makes
your body acidic. When your system becomes slightly acidic, free
radical formation and oxidative reactions increase. More serious
consequences of body's acidity is the fact that it causes calcium to be
mobilized out of your bones to buffer the acid and make your body more
alkaline. The calcium that is mobilized from your bone to neutralized
the acidity is then lost in the urine. Body acidity is the major cause
for eotroporosis.


Cancer Chemoprevention in Trout, Rodents, and Human Populations:
Studies by Drs. Bailey, Hendricks, and Pereira have shown that dietary
co-treatment of AFB1-exposed trout with the chlorophyll derivative
Cu-chlorophyllin (CHL) strongly reduces hepatic tumor initiation.
Molecular dosimetry in the trout revealed that reduction in DNA adducts
was a quantitatively predictive biomarker for tumor reduction under
certain conditions. Mechanistic studies in the trout (Bailey) and rat
(Dashwood) models support a very simple chemoprotective mechanism in
which strong non-covalent CHL-carcinogen complex formation reduced
systemic carcinogen uptake. Dr. Bailey is now collaborating with
Groopman and Kensler at the Johns Hopkins NIEHS Center on their
NIEHS-funded clinical intervention trial in Daxin, China, to assess CHL
effects on biomarkers of human AFB1 dietary exposure. This
collaboration combines the strengths of two NIEHS Centers to translate
knowledge first gained in aquatic models directly into the human
prevention setting. Dr. Dashwood's studies suggest equally promising
CHL protection against colon carcinogenesis in rats by co-exposure with
dietary heterocyclic amines, and a 13,000 trout chemoprevention
experiment with Drs. Bailey, Hendricks, and Pereira has shown strong,
dose-responsive CHL protection against all tumor phenotypes in the
multi-organ trout DBP model.




  • Oregon State University Environmental Health Sciences Center - Carcinogenesis Core







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